I dont buy this, anecdotal evidence isnt good enough when it comes to mental health dangers. They need to promote the studies showing no link. Or leave legalisation alone
Sir Niall, if you meant that there should be no legalisation of cannabis, until it can be proven that no causal linkage be show between cannabis usage and mental illenss, then I’ll address your quote with two points:
(otherwise ignore the two points immediately below)
1) Agreed. Scientific evidence is required. However, all reputable science has shown that no such causal link exists between cannabis use and mental illness. In this case, the problem with proving a negative, is that all methodologically sound good studies will show no link exists. Hence, the burden of proof lies with those trying to prove such a causal linkage exists between cannabis use and mental health damage. So far, NONE has been proven.
2) If the requirement for legalisation, of any substance, was to show no causal link between mental illness (or any other kind of illness) and that substance, then booze, tobacco, prescription drugs, non-prescription drugs, health substances, fast food, sweets, doughnuts, and so on would be illegal. That bar is way too high and it would be nearly impossible to criminalise all the substances mentioned previously. Prhohibition has been shown, time and again, to not work!
Since Professor Robin Murray‘s study is not yet published, I have not yet read it. Thus, currently, I can not give an informed opinion of it.
ASIDE
Most studies of this sort are, in my opinion as a research scientist, usually seriously flawed in terms of methodology. Unfortunately, these studies are also often funded by state agencies that, in many cases, have been taken over by political hacks instead of scientists. Hence, their agenda gets hijacked from being scientific research centres into being propaganda centres. (cf any studies done by the American NIMH or NIH, under the neo-cons. Under George Bush, these agencies only got funding for research that towed the government’s line. Naysayers were fired or blackballed. The Nutt firing, worried me that British research will soon follow the American path.)
I have not posted in a while, because I am crazy busy, but when I see shit like "cannabis makes you crazy" (Jesus we have gone back to the "Reffer Madness" propaganda of the 1930's amd 1940's) it should be refuted.
However, I will not have time to reply to this post.
I will not go into a detailed critique of Murray‘s study when it comes out, nor will I do so here, but I will leave you with some food for thought. (I am, and will be, for the next 10 months kookoo busy = very little time online and even less time to do thorough peer reviewing of research.)
If Murray‘s methodology consisted of
A) Separating participants into two camps:
1) a group of psychiatric patients (or those who have had a psychotic event) and
2) a control group (i.e. never had psychiatric care nor psychotic episodes),
B) then examining their historic cannabis use (including, the quantities) and comparing between the groups.
C) Then drawing conclusions regarding any causality, due to cannabis, from these steps
Then, from a statistical validity point of view, the study is completely bogus!
This is due to huge flaws in the methodology.
Some issues to consider are the following:
1) These groups have NOT been randomly selected. This would is a big NO-NO in research methodology.
2) Each groups’ members could have consumed a wide variety of substances other than (or including) cannabis. How do they account for the amount and effects of alcohol and other licit and illicit drugs consumed by the members?
3) There is no doubt* that psychiatric patients “self-medicate”, with both alcohol and drugs.
Hence it is NOT uncommon to see both higher rates of alcoholism and drug abuse in psychiatric patients. However, these folks are using substances AFTER they had mental illness. Nothing can be concluded about the causal attribution of those substances..
(*Anyone who has worked with either addicts or psychiatric patients or both will confirm this. As will the research in the respectable journals.)
Many times a persons will have been mentally ill for some time, but be “below that radar” because they have not yet a reported psychotic event. So, they continue to drink/toke/use other substances, until one day…BANG their illness has progressed to the point where they do have a psychotic event.
The emergency staff/psychiatrist/researcher might, then, wrongly, conclude that the illness was caused by the substance, since this patient had no history of mental illness before known substance use. (The patient simply had no REPORTED history of mental illness, but they were ill for months or years and it took time for their illness to erupt into a big enough event to be on the radar.
This also illustrates another problem associated with lumping the study participants into the two groups, as above. If a person with mild (or non-reported) mental illness, such as depression is put into the control group, then what happens if/when an illness occurs? Also if this person’s illness never gets reported and they have a low or no cannabis use for the duration of the study, then this person would be counted as a non-user with no mental illness. Obviously, this is incorrect!
4) Correlation does NOT IMPLY causation!!!!
Simply because a correlation (or “pattern” ) can be found among two or more variables does NOT mean that one causes the other. Nor would it tell you the direction of that relationship. (That is it, if there is a correlation between A and B, it does not imply that A causes B, nor that B causes A).
REMEMBER this Mantra every time you read about correlations:
Correlation does NOT IMPLY causation!!!!
Now consider the following:
If they did a longitudinal study which divided the participants into two groups
1) that used cannabis, and
2) that did not use cannabis
And then followed these groups for many years (after all this is a longitudinal study) and compared levels of psychotic events/psychiatric illness.
Then, this methodology would also present some big challenges, in addition to some of the same ones mentioned above.
Consider a member of group 2) (non cannabis users) who, at the outset of the study, had never used cannabis. What if, at say 5 years into the study they decide to try cannabis? Do you eliminate them from the study? Do you move them into the other group? Both of those options really cast doubt on any validity of the research.
When Murray‘s study is published, I hope that someone else on this board, with sufficient knowledge of both statistical and research methodology will crtique the study and post their opinions of it.
- Later